Suchbegriffe: VENTRICULAR REMODELING, . Treffer: 18
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CaMKII and PKA-dependent phosphorylation co-regulate nuclear localization of HDAC4 in adult cardiomyocytes.
Basic Res Cardiol. 2021; 116(1):11-11
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Ferrini, A; Stevens, MM; Sattler, S; Rosenthal, N
Toward Regeneration of the Heart: Bioengineering Strategies for Immunomodulation.
Front Cardiovasc Med. 2019; 6: 26
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Gonçalves, IF; Acar, E; Costantino, S; Szabo, PL; Hamza, O; Tretter, EV; Klein, KU; Trojanek, S; Abraham, D; Paneni, F; Hallström, S; Kiss, A; Podesser, BK
Epigenetic modulation of tenascin C in the heart: implications on myocardial ischemia, hypertrophy and metabolism.
J Hypertens. 2019; 37(9):1861-1870
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Koentges, C; Pepin, ME; Müsse, C; Pfeil, K; Alvarez, SVV; Hoppe, N; Hoffmann, MM; Odening, KE; Sossalla, S; Zirlik, A; Hein, L; Bode, C; Wende, AR; Bugger, H
Gene expression analysis to identify mechanisms underlying heart failure susceptibility in mice and humans.
Basic Res Cardiol. 2018; 113(1):8-8
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Sharp, TE; Schena, GJ; Hobby, AR; Starosta, T; Berretta, RM; Wallner, M; Borghetti, G; Gross, P; Yu, D; Johnson, J; Feldsott, E; Trappanese, DM; Toib, A; Rabinowitz, JE; George, JC; Kubo, H; Mohsin, S; Houser, SR
Cortical Bone Stem Cell Therapy Preserves Cardiac Structure and Function After Myocardial Infarction.
Circ Res. 2017; 121(11):1263-1278
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Toth, GG; Vanderheyden, M; Bartunek, J
Novel device-based interventional strategies for advanced heart failure.
Postepy Kardiol Interwencyjnej. 2016; 12(1):13-16
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Beer, L; Zimmermann, M; Mitterbauer, A; Ellinger, A; Gruber, F; Narzt, MS; Zellner, M; Gyöngyösi, M; Madlener, S; Simader, E; Gabriel, C; Mildner, M; Ankersmit, HJ
Analysis of the Secretome of Apoptotic Peripheral Blood Mononuclear Cells: Impact of Released Proteins and Exosomes for Tissue Regeneration.
Sci Rep. 2015; 5(44):16662-16662
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Hohendanner, F; Ljubojević, S; MacQuaide, N; Sacherer, M; Sedej, S; Biesmans, L; Wakula, P; Platzer, D; Sokolow, S; Herchuelz, A; Antoons, G; Sipido, K; Pieske, B; Heinzel, FR
Intracellular dyssynchrony of diastolic cytosolic [Ca²⁺] decay in ventricular cardiomyocytes in cardiac remodeling and human heart failure.
Circ Res. 2013; 113(5):527-538
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Nagy, B; Nagaraj, C; Kwapiszewska, G; Olschewski, A; Olschewski, H
Right Ventricular Diastolic Dysfunction in ABCG2 knockout mice under Hypoxia
CIRCULATION. 2013; 128(22):
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Bourgonje, VJ; Schoenmakers, M; Beekman, JD; van der Nagel, R; Houtman, MJ; Miedema, LF; Antoons, G; Sipido, K; de Windt, LJ; van Veen, TA; Vos, MA
Relevance of calmodulin/CaMKII activation for arrhythmogenesis in the AV block dog.
Heart Rhythm. 2012; 9(11):1875-1883
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Beholz, S; Repossini, A; Livi, U; Schepens, M; El Gabry, M; Matschke, K; Trivedi, U; Eckel, L; Dapunt, O; Zamorano, JL
The Freedom SOLO valve for aortic valve replacement: clinical and hemodynamic results from a prospective multicenter trial.
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Beer, M; Machann, W; Sandstede, J; Buchner, S; Lipke, C; Kostler, H; Lorenz, R; Harre, K; Spindler, M; Hahn, D
Energetic differences between viable and non-viable myocardium in patients with recent myocardial infarction are not an effect of differences in wall thinning - a multivoxel P-31-MR-spectroscopy and MRI study
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Clin Chem. 2007; 53(4):773-780
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Nahrendorf, M; Hu, K; Frantz, S; Jaffer, FA; Tung, CH; Hiller, KH; Voll, S; Nordbeck, P; Sosnovik, D; Gattenlohner, S; Novikov, M; Dickneite, G; Reed, GL; Jakob, P; Rosenzweig, A; Bauer, WR; Weissleder, R; Ertl, G
Factor XIII deficiency causes cardiac rupture, impairs wound healing, and aggravates cardiac remodeling in mice with myocardial infarction.
Circulation. 2006; 113(9):1196-1202
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Watzinger, N; Lund, GK; Higgins, CB; Wendland, MF; Weinmann, HJ; Saeed, M
The potential of contrast-enhanced magnetic resonance imaging for predicting left ventricular remodeling.
J Magn Reson Imaging. 2002; 16(6):633-640
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Haghighi, K; Schmidt, AG; Hoit, BD; Brittsan, AG; Yatani, A; Lester, JW; Zhai, J; Kimura, Y; Dorn, GW; MacLennan, DH; Kranias, EG
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Huber, K; Pachinger, O; Pichler, M; Klein, W
Pathophysiological mechanisms of the renin-angiotensin system and its pharmacologic modification by ACE inhibitors or angiotensin II (type 1) receptor blockers in cardiovascular diseases
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