Medizinische Universität Graz Austria/Österreich - Forschungsportal - Medical University of Graz
Publikationen
Suchbegriffe: CNS INFLAMMATION, . Treffer: 9
2021
Runtsch, MC; Ferrara, G; Angiari, S
Metabolic determinants of leukocyte pathogenicity in neurological diseases.
J Neurochem. 2021; 158(1):36-58
Doi: 10.1111/jnc.15169
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2015
Zeka, B; Hastermann, M; Hochmeister, S; Kögl, N; Kaufmann, N; Schanda, K; Mader, S; Misu, T; Rommer, P; Fujihara, K; Illes, Z; Leutmezer, F; Sato, DK; Nakashima, I; Reindl, M; Lassmann, H; Bradl, M
Highly encephalitogenic aquaporin 4-specific T cells and NMO-IgG jointly orchestrate lesion location and tissue damage in the CNS.
Acta Neuropathol. 2015; 130(6):783-798
Doi: 10.1007/s00401-015-1501-5
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2014
Bosticardo, M; Musio, S; Fontana, E; Angiari, S; Draghici, E; Constantin, G; Poliani, PL; Pedotti, R; Villa, A
Development of central nervous system autoimmunity is impaired in the absence of Wiskott-Aldrich syndrome protein.
PLoS One. 2014; 9(1): e86942-e86942.
Doi: 10.1371/journal.pone.0086942
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2013
Adzemovic, MZ; Zeitelhofer, M; Hochmeister, S; Gustafsson, SA; Jagodic, M
Efficacy of vitamin D in treating multiple sclerosis-like neuroinflammation depends on developmental stage.
Exp Neurol. 2013; 249(12):39-48
Doi: 10.1016/j.expneurol.2013.08.002
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Kitic, M; Hochmeister, S; Wimmer, I; Bauer, J; Misu, T; Mader, S; Reindl, M; Fujihara, K; Lassmann, H; Bradl, M
Intrastriatal injection of interleukin-1 beta triggers the formation of neuromyelitis optica-like lesions in NMO-IgG seropositive rats.
Acta Neuropathol Commun. 2013; 1(11):5-5
Doi: 10.1186/2051-5960-1-5
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2011
Rossi, B; Zenaro, E; Angiari, S; Ottoboni, L; Bach, S; Piccio, L; Pietronigro, EC; Scarpini, E; Fusco, M; Leon, A; Constantin, G
Inverse agonism of cannabinoid CB1 receptor blocks the adhesion of encephalitogenic T cells in inflamed brain venules by a protein kinase A-dependent mechanism.
J Neuroimmunol. 2011; 233(1-2): 97-105.
Doi: 10.1016/j.jneuroim.2010.12.005
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2009
Hochmeister, S; Bittner, RE; Höger, H; Lassmann, H; Bradl, M
The susceptibility to experimental autoimmune encephalomyelitis is not related to dysferlin-deficiency.
Autoimmunity. 2009; 42(3): 235-241.
Doi: 10.1080/08916930802716542
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Schoderboeck, L; Adzemovic, M; Nicolussi, EM; Crupinschi, C; Hochmeister, S; Fischer, MT; Lassmann, H; Bradl, M
The "window of susceptibility" for inflammation in the immature central nervous system is characterized by a leaky blood-brain barrier and the local expression of inflammatory chemokines.
Neurobiol Dis. 2009; 35(3): 368-375.
Doi: 10.1016/j.nbd.2009.05.026
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2003
Weissert, R; Wiendl, H; Pfrommer, H; Storch, MK; Schreiner, B; Barth, S; Seifert, T; Melms, A; Dichgans, J; Weller, M
Action of treosulfan in myelin-oligodendrocyte-glycoprotein-induced experimental autoimmune encephalomyelitis and human lymphocytes.
J NEUROIMMUNOL 2003 144: 28-37.
Doi: 10.1016%2Fj.jneuroim.2003.08.028
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